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In the matrix, the metallo-β-lactamase-family RNase ELAC2 requires zinc for tRNA 3′-end processing, a key step for mitochondrial translation and the assembly of respiratory complexes 85
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Given GSH’s known direct protective role in mitochondrial metabolism and its association with chronic diseases of highly metabolically active tissues, this review aims to examine the literature for evidence that low GSH levels may be an important causative factor in the development of chronic illnesses
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