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This figure is modified from Klessig ( In addition to GAPDH’s role in viral infection, it is a major suspect in several neurodegenerative diseases in humans, including Huntington’s, Parkinson’s, and Alzheimer’s diseases ( in absentia homolog (Siah) ( in vitro and in vivo models, prevents S-nitrosylation of GAPDH, blocks the GAPDH–Siah interaction, and inhibits GAPDH nuclear translocation ( Using recombinant human GAPDH, we demonstrated that SA not only binds this protein but also suppresses its ability to translocate to the nucleus and induce cell death at low micromolar concentrations ( G