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Oxidative stress plays a crucial role in the development of age-related diseases including arthritis, diabetes, dementia, cancer, atherosclerosis, vascular diseases, obesity, osteoporosis, and metabolic syndromes (Tan et al., 2015a
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Within this framework, the ACOD1/IRG1-itaconate axis functions as an inflammatory metabolic brake that shapes the fate of macrophages, dendritic cells, and T cells
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