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An HD rat model accurately mimics GSTO2 overexpression The previously unappreciated causal link between altered glutathione–ascorbate metabolism and changes in dopaminergic signalling upon loss of BDNF-TrkB signalling prompted us to investigate its relevance to HD pathogenesis
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Further studies have shown that hyperuricemia can accelerate glomerular hypertension and the vascular lesions, resulting in worsening proteinuria and renal dysfunction [60]
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